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These terms, which are used interchangeably, refer to a tremor that is present when the limbs and trunk are actively maintained in certain positions (such as holding the arms outstretched) and that may persist throughout active movement More particularly, the tremor is absent when the limbs are relaxed but becomes evident when the muscles are activated It is accentuated as greater precision of movement is demanded, but it does not approach the degree of augmentation seen with so-called intention tremor, which is associated with cerebellar disease In contrast to rest or static (parkinsonian) tremor, which is characterized electromyographically by alternating activity in agonist and antagonist muscles, most cases of action tremor are characterized by relatively rhythmic bursts of grouped motor neuron discharges that occur not quite synchronously and simultaneously in opposing muscle groups Slight inequalities in the strength and timing of contraction of opposing muscle groups account for the tremor Action tremors are of several different types, a feature that makes them more dif cult to interpret than other tremors Enhanced Physiologic Tremor One type of action tremor seems merely to be an exaggeration of normal or physiologic tremor; it has the same fast frequency as physiologic tremor (about 10 Hz) but a greater amplitude Such a tremor, best elicited by holding the arms outstretched with ngers spread apart, is characteristic of intense fright and anxiety, certain metabolic disturbances (hyperthyroidism, hypercortisolism, hypoglycemia), pheochromocytoma, intense physical exertion, withdrawal from alcohol and other sedative drugs, and the toxic effects of several drugs lithium, nicotinic acid, xanthines (coffee, tea, aminophylline, colas), and corticosteroids It is noteworthy that a transient action tremor of this type can be reproduced by the intravenous injection of epinephrine or beta-adrenergic drugs such as isoproterenol Young and colleagues have determined that the enhancement of physiologic tremor that occurs in these various metabolic and toxic states is due to stimulation of certain muscular tremorogenic beta-adrenergic receptors by increased levels of circulating catecholamines Thus it appears that synchronization of motor units in physiologic tremor, though not primarily of neural origin, is nevertheless in uenced by central and peripheral nervous activity Alcohol Withdrawal Tremor The most prominent feature of the alcohol withdrawal syndrome is a special type of action tremor 80.

The Properties screen area provides access to all the properties of the selected Web item or the Web template itself The component you select determines the number of properties associated with the Web item Also, all the parameters and settings are configured here The Properties screen area uses two tabs to segment the properties The General tab, shown next, provides access to the data provider assignment, if required The Web Item Parameters is, in most cases, the more important portion and shows a listing of the configuration settings of that component The Properties screen area is very important to developers and is where you will spend most of your time configuring the WAD template

rho, 33 theta, 32 vega, 32 33 H Hard stops, 80, 85 monetary, 80 price, 81 Hedge funds, 1 3 Hedging: by commodities traders, 31 with interest-rate options, 244 Historical volatility, 37 38 Horizontal spreads, 115 I Implied volatility (IV), 38 39 on credit spreads, 166 on iron butter y spreads, 202 203 for long straddles, 169 and pricing of options, 254 259 and selling of options, 90 94 with short call positions, 101 102 in short delta-neutral trading, 185 186 and straddles or strangles, 166 167 Implied volatility stops, 83 84 Index funds, 1 4, 216 217 (See also Exchange-traded funds)

Posttraumatic Epilepsy Epilepsy is the most common delayed sequela of craniocerebral trauma, with an overall incidence of about 5 percent in patients with closed head injuries and 50 percent in those who had sustained a compound skull fracture and wound of the brain The basis is nearly always a contusion or laceration of the cortex As one might expect, the risk of developing posttraumatic epilepsy is also related to the overall severity of the closed head injury In a civilian cohort of 2747 head-injured patients described by Annegers and colleagues, the risk of seizures after severe head injury (loss of consciousness or amnesia for more than 24 h, including subdural hematoma and brain contusion) was 7 percent within 1 year and 115 percent in 5 years If the injury was only moderate (unconsciousness or amnesia for 30 min to 24 h or caus-

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